Author

Nguyen Le Quang

Oxford University Clinical Research UnitORCID 0000-0002-0083-8896Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Microbiology· 3

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:human· 2 Approach:statistical model· 2 Organism:bacteria (other)· 1 Assay:bulk RNA-seq· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Nguyễn Thụy Thương ThươngiD· 3 shared Đặng Thị Minh Hà· 2 shared Do Dang Anh Thu· 2 shared Maxine CawsiD· 2 shared Phan Vuong Khac Thai· 2 shared A. N. HalliD· 1 shared Abigail M FreyiD· 1 shared Babak JavidiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersMicrobiology

Nguyễn Thụy Thương ThươngORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

MicrobiologybioRxivPreprintApr 8, 2026

Long-read sequencing of Mycobacterial tuberculosis is comparable to short-read sequencing for antimicrobial resistance prediction and epidemiological studies.

Matthew Colpus, Catriona S Baker, Eloïse Roghi…·Derrick W. Crook

This study compares long-read and short-read sequencing for tuberculosis drug resistance and outbreak analysis, finding they are comparable with minimal differences in key clinical outputs.

1 citation

humanwhole-genome sequencing

𝕏 PostBluesky

MicrobiologyCellPeer-reviewedSep 30, 2025

Evolution of Mycobacterium tuberculosis transcription regulation is associated with increased transmission and drug resistance

Peter H. Culviner, Abigail M Frey, Qingyun Liu…·Sarah M. Fortune

The paper identifies variants in Mycobacterium tuberculosis associated with decreased virulence gene expression and increased transmissibility, particularly in drug-resistant strains.

25 citationstop 1%

bacteria (other)bulk RNA-seq

𝕏 PostBluesky

MicrobiologyeLifePeer-reviewedFeb 2, 2024

Rifampicin tolerance and growth fitness among isoniazid-resistant clinical Mycobacterium tuberculosis isolates: an in-vitro longitudinal study

Srinivasan Vijay, Nguyen Le Hoai Bao, Dao Nguyen Vinh…·Nguyễn Thụy Thương Thương

This study examines rifampicin tolerance in isoniazid-resistant Mycobacterium tuberculosis isolates, finding increased tolerance and faster growth in resistant strains, which may contribute to multidrug-resistant TB emergence.

6 citations

humanstatistical model

𝕏 PostBluesky