Author

Jakob Troppmair

Innsbruck Medical UniversityORCID 0000-0002-0611-3837Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Organism:mouse· 1 Assay:protein structure· 1

Frequent Co-Authors

Andreas FeichtneriD· 1 shared Christine Doblander· 1 shared Eduard StefaniD· 1 shared Erich GnaigeriD· 1 shared Florian EnzleriD· 1 shared Hélène LemieuxiD· 1 shared Jakob Fleischmann· 1 shared Martin Wurm· 1 shared

Activity by Year

2017-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperBiochemistry

Andreas FeichtnerORCID

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2 papers

Recent·Most cited

BiochemistryeLifePeer-reviewedFeb 20, 2024

Kinases in motion: impact of protein and small molecule interactions on kinase conformations

Valentina Kugler, Selina Schwaighofer, Andreas Feichtner…·Eduard Stefan

This study uses KinCon-reporter technology to analyze conformational changes in disease-associated kinases upon drug, regulatory protein, and pathway activation interactions, revealing insights into therapeutic strategies.

6 citations

humanprotein structure

𝕏 PostBluesky

Cancer BiologybioRxivPreprintJun 19, 2017

Impairment of mitochondrial respiratory function as an early biomarker of apoptosis induced by growth factor removal

Hélène Lemieux, Patrick Subarsky, Christine Doblander…·Erich Gnaiger

The paper shows that removal of IL-3 impairs mitochondrial respiratory function in 32D cells, with v-RAF expression mitigating this effect.

DOI 10.1101/151480

mouseexperimental assay

𝕏 PostBluesky