Author

Hermann Broder Schmidt

Stanford MedicineORCID 0000-0002-0750-8419Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:human· 3 Research Focus:cell signaling & trafficking· 2 Assay:proteomics· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1

Frequent Co-Authors

Aaron D. GitleriD· 1 shared Adam BrineriD· 1 shared Alex S. HolehouseiD· 1 shared Ariana Barreau· 1 shared Beryl B. CummingsiD· 1 shared Björn OskarssoniD· 1 shared Bradley F. BoeveiD· 1 shared Caitlin M. RodrigueziD· 1 shared

Activity by Year

2019-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 28 mutual co-author links

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1 shared paperNeuroscience

Aaron D. GitlerORCID

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3 papers

Recent·Most cited

Cell BiologybioRxivPreprintMar 17, 2026

C9ORF72-derived polyGR polypeptides disrupt passive nucleocytoplasmic transport by tuning protein affinity for the nuclear pore barrier

Daniel A. Solomon, Ryan J. Emenecker, Marie-Therese Salcher-Konrad…·Sarah Mizielinska

This study shows how polyGR polypeptides from C9ORF72 disrupt nucleocytoplasmic transport by altering protein affinity for the nuclear pore FG barrier, leading to biphasic transport outcomes and protein aggregation.

1 citation

humanproteomics

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NeuroscienceNaturePeer-reviewedFeb 23, 2022

TDP-43 represses cryptic exon inclusion in FTD/ALS gene UNC13A

X. Rosa Ma, Mercedes Prudencio, Yuka Mitsuhashi Koike…·Aaron D. Gitler

This study shows that TDP-43 represses a cryptic exon in UNC13A, and genetic variants associated with FTD/ALS increase cryptic exon inclusion when TDP-43 is dysfunctional.

574 citationstop 1%

humanbulk RNA-seq

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Cell BiologybioRxivPreprintFeb 13, 2019

Decoding and recoding phase behavior of TDP43 reveals that phase separation is not required for splicing function

Hermann Broder Schmidt, Ariana Barreau, Rajat Rohatgi

The paper shows that TDP43's splicing function does not require phase separation, using mutagenesis and live-cell assays to analyze phase dynamics.

DOI 10.1101/548339

humanproteomics

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