Author

Alhafidz Hamdan

Edinburgh Cancer ResearchORCID 0000-0002-0794-5504Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Alhafidz Hamdan1 paper · Institute of Genetics and Cancer;

Biology Disciplines

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Cancer Biology· 1 Genetics· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Assay:imaging· 1 Research Focus:protein structure prediction· 1

Frequent Co-Authors

Ailith EwingiD· 1 shared Alison M. MeynertiD· 1 shared Andrew Victor BiankiniD· 1 shared Athena MatakidouiD· 1 shared Brian Andrew DoughertyiD· 1 shared Brian McDade· 1 shared C. Simon HerringtoniD· 1 shared Charlie GourleyiD· 1 shared

Activity by Year

2022-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCancer Biology

Ailith EwingORCID

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2 papers

Recent·Most cited

Cancer BiologyNature CommunicationsPeer-reviewedJul 1, 2025

Divergent trajectories to structural diversity impact patient survival in high grade serous ovarian cancer

Ailith Ewing, Alison M. Meynert, Ryan Silk…·Colin A M Semple

The paper identifies two divergent structural variation trajectories in HGSOC that impact patient survival and reveals novel candidate driver genes and mtDNA mutations associated with prognosis.

19 citationstop 3%

humanwhole-genome sequencing

𝕏 PostBluesky

GeneticseLifePeer-reviewedOct 28, 2022

Oncogene expression from extrachromosomal DNA is driven by copy number amplification and does not require spatial clustering

Karin R. Purshouse, Elias T. Friman, Shelagh Boyle…·Wendy A. Bickmore

The paper investigates oncogene expression from extrachromosomal DNA in glioblastoma and finds that copy number amplification, not spatial clustering, drives high transcriptional output.

61 citationstop 1%

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