Jessica N. Sowa

This study identifies Mucor hiemalis as a pathogen of Caenorhabditis nematodes, showing it kills hosts via intestinal perforation and does not trigger typical immune responses.

This study examines diversity-disease relationships in wild nematode communities, finding that species-poor communities have higher microsporidia prevalence and immune activation.

The paper identifies specific bacterial metabolites affecting C. elegans physiology and develops an RNA interference strain to study genotype-phenotype interactions without microbial confounding.

This study identifies a novel pathway, the Intracellular Pathogen Response (IPR), that promotes proteostasis in C. elegans by upregulating ubiquitin ligase components like cul-6, with pals-22 acting as a repressor.