Paola Fabrizio

This study shows that loss of SIN-3 in C. elegans disrupts mitochondrial function and metabolism, leading to mitochondrial fragmentation and altered metabolic flux.

This study shows that reduced H3K4 methylation in C. elegans shortens lifespan and reduces fertility, contradicting previous findings that suggested H3K4 methylation extends lifespan.

The paper shows that auxin treatment in C. elegans activates the UPR to confer resistance to ER stress, highlighting potential confounding effects of auxin in studies using the AID system.