NeurosciencePLoS Computational BiologyPeer-reviewed
A multi-state model of the CaMKII dodecamer suggests a role for calmodulin in maintenance of autophosphorylation
Matthew C Pharris, Neal M. Patel, Tyler G. VanDyk…·Tamara L. Kinzer‐Ursem
The paper develops a rule-based computational model of CaMKII holoenzyme to explore how Ca2+/CaM binding stabilizes autophosphorylation and regulates synaptic plasticity.
24 citations