David Goich

This study identifies Yck2's role in linking mitochondrial function, pH homeostasis, and cell wall remodeling in Cryptococcus neoformans, revealing mechanisms behind its impact on antifungal susceptibility.

This study investigates the role of eIF3 subunits in stress adaptation and pathogenesis of C. neoformans, showing sensitivity to mitochondrial stress and differential drug responses in specific mutants.

The paper investigates the role of Hog1/p38 and Gcn2 pathways in C. neoformans' response to oxidative stress, showing their convergence on transcriptomic changes and stress tolerance.

This study shows that Gcn2 compensates for Hog1/p38 loss in C. neoformans during thermal stress, rescuing adaptation and virulence through crosstalk between these pathways.

This study examines how Ccr4 and Gcn2 pathways contribute to translational reprogramming in C. neoformans under host-relevant stressors, showing differential roles in oxidative stress and temperature stress.