Michael J. Holmes

This paper shows that GCN5a loss in Toxoplasma gondii promotes latency by causing telomere dysfunction and altering gene expression during differentiation.

This study identifies cap-independent translation as a mechanism driving stress-induced differentiation in Toxoplasma gondii by regulating BFD1 protein synthesis.

This study identifies eIF1.2 as a regulator of Toxoplasma gondii differentiation by controlling the translation of key bradyzoite factors.

This study identifies eIF4E1 as a key regulator of Toxoplasma gondii latency by showing its role in translational control during encystation.