Robert E. White

The paper uses nanopore sequencing to show herpesvirus mRNAs have longer poly(A) tails than cellular or other viral transcripts, suggesting a unique mechanism for enhancing gene expression.

This paper uses long-read RNA-seq to analyze EBV transcriptomes in LCLs, identifying new splicing variants, polyA sites, and transcript structures, and provides tools and annotations for future studies.

The paper identifies Sp140L as a novel restriction factor that herpesviruses counteract to evade innate immune responses and establish infection.

This study shows virus-induced paraspeckle-like structures are critical for viral replication and contribute to genomic instability during KSHV and EBV infections.

This study identifies EBNA-LP's role in promoting naive B cell transformation by interacting with YY1 through conserved leucine-rich motifs.