Author

Mridushi Daga

University of Massachusetts Chan Medical SchoolORCID 0000-0002-5120-8720Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Immunology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell-cell communication· 2 Organism:human· 2 Organism:mouse· 2 Assay:spatial transcriptomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Ann Marshak‐RothsteiniD· 2 shared Christina E. BaeriD· 2 shared Haya S. Raef· 2 shared Jillian M. RichmondiD· 2 shared Khashayar AfshariiD· 2 shared Mehdi RashighiiD· 2 shared Nazgol‐Sadat HaddadiiD· 2 shared Robert Li· 2 shared

Activity by Year

2026-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersImmunology

Ann Marshak‐RothsteinORCID

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3 papers

Recent·Most cited

ImmunologyNature CommunicationsPeer-reviewedJul 27, 2026

Comparative spatial transcriptomics of hair follicle-T cell interactions in mouse, dog and human reveals conserved drivers of primary cicatricial alopecia

Ümmügülsüm Yıldız-Altay, Rutha Adhanom, Warda Abdi…·Jillian M. Richmond

This study uses spatial transcriptomics across mouse, dog, and human to identify conserved T cell-hair follicle interactions in PCA, revealing potential drug targets.

DOI 10.1038/s41467-026-76048-8

mousehuman

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ImmunologyScientific ReportsPeer-reviewedJul 22, 2026

Spatial characterization of interface dermatitis in cutaneous lupus reveals novel chemokine ligand-receptor pairs that drive disease

Saeed Shakiba, Nazgol‐Sadat Haddadi, Khashayar Afshari…·Jillian M. Richmond

This study identifies novel chemokine ligand-receptor pairs involved in immune cell recruitment in cutaneous lupus by combining spatial transcriptomics, proteomics, and immunological assays.

7 citationstop 1%

humanspatial transcriptomics

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ImmunologyJournal of Investigative DermatologyPeer-reviewedMar 28, 2026

Endothelial Cell Expression of STINGV154M Gain-of-Function Mutation Delays the Resolution of UVB-induced Skin Injury

Jane Chuprin, Carolina Salomão Lopes, Kevin MingJie Gao…·Mehdi Rashighi

This study shows that endothelial cells with a STINGV154M mutation cause persistent skin inflammation after UVB exposure, linking STING activation to impaired myeloid cell recruitment and delayed inflammation resolution.

DOI 10.1016/j.jid.2026.03.014

mousebulk RNA-seq

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