Author

Guido Zagnoli-Vieira

University of CambridgeORCID 0000-0002-5656-1056Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:human· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

Christopher J. CarnieiD· 2 shared Petra BeliiD· 2 shared Samah W. AwwadiD· 2 shared Simon LamiD· 2 shared Stephen P. JacksoniD· 2 shared Aldo S. BaderiD· 1 shared Almudena Serrano-BeníteziD· 1 shared Amy R. Wanders· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersGenetics

Christopher J. CarnieORCID

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2 papers

Recent·Most cited

GeneticsEMBO Molecular MedicinePeer-reviewedDec 19, 2025

Loss of CTLH component MAEA impairs DNA repair and replication and leads to developmental delay

Soren H. Hough, Satpal S. Jhujh, Samah W. Awwad…·Stephen P. Jackson

The paper identifies MAEA as a critical regulator of DNA repair and replication, with loss of function causing developmental delay and neurodevelopmental deficits.

2 citations

humanDNA methylation

𝕏 PostBluesky

Cell BiologyThe EMBO JournalPeer-reviewedMay 17, 2024

The dCMP deaminase DCTD and the E3 ligase TOPORS are central mediators of decitabine cytotoxicity

Christopher J. Carnie, Maximilian J. Götz, Chloe S. Palma-Chaundler…·Stephen P. Jackson

This study identifies DCTD and TOPORS as key factors in decitabine cytotoxicity by showing their roles in DPC formation and repair.

41 citationstop 3%

humangenetic perturbation / CRISPR

𝕏 PostBluesky