Author

Eitaro Kume

Kyoto UniversityORCID 0000-0002-8913-6814Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

Akira KakizukaiD· 1 shared Chiharu OtaniiD· 1 shared Daisuke YoshinagaiD· 1 shared Fumiaki Fukumura· 1 shared Fuquan Zou· 1 shared Hidenori KojimaiD· 1 shared Hiroaki FujitaiD· 1 shared Hirohito KubotaiD· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperPathology

Akira KakizukaORCID

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2 papers

Recent·Most cited

PathologyAtherosclerosisPeer-reviewedMay 1, 2025

A novel VCP modulator, KUS121, attenuates atherosclerosis progression by maintaining intracellular ATP and mitigating ER stress in endothelial cells

Fuquan Zou, Osamu Baba, Takahiro Horie…·Koh Ono

This study shows that KUS121 reduces atherosclerosis by maintaining ATP levels and reducing ER stress in endothelial cells.

2 citations

mouseproteomics

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Cell BiologyScientific ReportsPeer-reviewedApr 17, 2024

HOIL-1L deficiency induces cell cycle alteration which causes immaturity of myocyte and fibrogenesis

Kentaro Akagi, Shiro Baba, Hiroaki Fujita…·Junko Takita

This study shows HOIL-1L deficiency leads to cell cycle defects and fibrosis in myotubes and cardiomyocytes, contributing to myopathy and dilated cardiomyopathy.

DOI 10.1038/s41598-024-57504-1

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