Ricardo Martin Vidal

This study shows that the NSP4 T492I mutation increases SARS-CoV-2 mutation rates and accelerates the emergence of Omicron-like mutations.

This study investigates how tyrosine-based sorting signals in the ORF3a protein of SARS-CoV-2 affect intracellular trafficking, apoptosis, and autophagy, finding that multiple motifs are required for efficient transport and that surface expression is not necessary for autophagy.

The paper uses hamster sera from variant-specific infections to map antigenic differences among SARS-CoV-2 variants, revealing significant antigenic variation among Omicron subvariants.