Author

Christian Stockmann

University of ZurichORCID 0000-0002-9726-6838Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Adrián Salas-BastosiD· 1 shared Amandine FrancoisiD· 1 shared Ariunbold Chuluun-ErdeneiD· 1 shared Atsumasa KurozumiiD· 1 shared Claire BardetiD· 1 shared Daigo SawakiiD· 1 shared Fanny CollaudiD· 1 shared Ganesh PathareiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperPhysiology

Adrián Salas-BastosORCID

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2 papers

Recent·Most cited

Cell BiologybioRxivPreprintJan 30, 2026

The REFLEX system enables in vivo identification of perivascular angiogenic macrophages in the heart

Tatsuyuki Sato, Takayuki Isagawa, Toshinaru Kawakami…·Norihiko Takeda

The paper develops REFLEX and HUNTERuni-seq to identify perivascular angiogenic macrophages (PVAMs) that deliver VEGF-A to endothelial cells, showing physical contact is critical for effective signaling.

DOI 10.64898/2026.01.27.702189

mousescRNA-seq

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PhysiologybioRxivPreprintJan 18, 2024

Chronically elevated FGF23 drives sustained renal ERK signaling and inflammatory transcriptional programs mitigated by cFGF23 gene therapy

Adrián Salas-Bastos, Claire Bardet, Klaudia Kopper…·Ganesh Pathare

This study shows that chronic FGF23 elevation causes kidney inflammation via sustained ERK signaling, which is reduced by cFGF23 gene therapy.

3 citations

mouseexperimental assay

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