Author

Anja K. Münster-Kühnel

Medizinische Hochschule HannoverORCID 0000-0003-0991-7627Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Assay:proteomics· 2 Research Focus:benchmarking· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1

Frequent Co-Authors

Alexandre StellaiD· 1 shared Ann-Kristin JochumiD· 1 shared Anna C. ObenaufiD· 1 shared Anne Gonzalez de PeredoiD· 1 shared Aurélien BoyanceiD· 1 shared Camille GorltiD· 1 shared David HoffmanniD· 1 shared Denis HudrisieriD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperImmunology

Alexandre StellaORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Cancer BiologyNature CommunicationsPeer-reviewedMay 22, 2026

Tumor sialylation controls effective anti-cancer immunity in breast cancer

Stefan Mereiter, Gustav Jonsson, Tiago Oliveira…·Josef Martin Penninger

This study shows that tumor sialylation suppresses anti-cancer immunity in breast cancer by altering immune cell interactions and checkpoint ligands, and that inhibiting sialylation enhances immune response and therapy effectiveness.

4 citations

humanproteomics

𝕏 PostBluesky

ImmunologybioRxivPreprintJan 12, 2026

ST8SIA4-mediated polysialylation is critical for CCR2-driven monocyte egress from the bone marrow

Aurélien Boyance, Nelly Gilles, Maxime Rotival…·Yoann Rombouts

This study identifies ST8SIA4-mediated polysialylation as essential for CCR2-driven monocyte egress from the bone marrow, revealing a novel regulatory mechanism in immune cell trafficking.

DOI 10.64898/2026.01.09.698551

mouseproteomics

𝕏 PostBluesky