Milica Radisic

The paper investigates how epicardial extracellular vesicles affect gene expression during ischemia-reperfusion injury using a heart-on-a-chip model.

This study shows that galectin-1 modulates cell adhesions, caveolae, and vascular permeability in kidney endothelial cells through proteomic and phosphoproteomic analyses.

This paper introduces a heart-on-a-chip platform with ultrasoft mechanosensors to measure cell- and tissue-scale contractile stresses, showing that ECM composition affects contractility and drug responses.

This study shows that biomimetic fractal topography improves podocyte maturation in vitro by enhancing marker expression, cell polarity, and ECM remodeling via YAP signaling.

This study shows that a myosin ATPase inhibitor reverses HCM phenotypes in pediatric iPSC-cardiomyocytes, with effects dependent on the underlying genetic variant.

The paper introduces a MATLAB tool for quantitatively analyzing 2D tissue images, measuring morphology and co-localization parameters with flexibility across different tissue types.

This paper develops a heart-on-a-chip model to study immune-induced cardiac dysfunction, identifying mitochondrial DNA as a key factor and showing therapeutic potential of endothelial exosomes.