Author

Onnik Agbulut

Centre National de la Recherche ScientifiqueORCID 0000-0003-1923-8871Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Bioengineering· 1 Pathology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Organism:human· 1 Assay:organoid / primary culture· 1

Frequent Co-Authors

Pierre JoanneiD· 2 shared Alba MarcellaniD· 1 shared Alexis CanetteiD· 1 shared Anne‐Marie RodrigueziD· 1 shared Anthony BéhiniD· 1 shared Aurore L’honoréiD· 1 shared Cécile LefebvreiD· 1 shared Christophe HélaryiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersBioengineering

Pierre JoanneORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

BioengineeringBiomaterials SciencePeer-reviewedJan 1, 2026

Fibrotic-like collagen matrices as innovative 3D in vitro models for investigating the impact of pathological ECM on muscle regeneration in muscular dystrophies

Marie Camman, Naomi Nieswic, Maxime Mauviel…·Christophe Hélary

This paper develops fibrotic-like collagen matrices to study how pathological ECM affects muscle regeneration in muscular dystrophies, finding that fibrotic matrices impair myoblast behavior.

1 citation

organoid / primary cultureexperimental assay

𝕏 PostBluesky

PathologyStem Cell Research & TherapyPeer-reviewedJan 2, 2024

Critical contribution of mitochondria in the development of cardiomyopathy linked to desmin mutation

Yeranuhi Hovhannisyan, Zhenlin Li, Domitille Callon…·Onnik Agbulut

This study shows that mitochondrial defects in desmin-mutated cardiomyocytes contribute to cardiomyopathy and can be reversed by transferring normal mitochondria.

34 citationstop 5%

humanbulk RNA-seq

𝕏 PostBluesky