Mercedes Pascual

The paper shows that higher-order interactions in ecosystems lead to abrupt community collapse rather than gradual extinction, challenging traditional pairwise theory.

This study shows that humidity modifies the thermal performance of key fitness traits in Anopheles stephensi, altering population fitness projections compared to temperature-only models.

The paper models how immune and functional selection shape multigene families in Plasmodium falciparum under varying transmission conditions, showing immune selection favors fast-recombining genes and reduces fitness variation.

The paper examines how host competitive asymmetries affect viral evolution in a co-evolutionary system, finding that they extend viral extinction times and accelerate adaptation.

The paper shows that relative humidity systematically shifts the temperature dependence of population growth in Anopheles stephensi, a malaria vector.

The paper examines the population structure of DBLα types in Sub-Saharan Africa, finding varying conservation levels across geographical scales and frequencies.

The paper uses a computational model to show that negative frequency-dependent selection contributes to the modular structure of effector repertoires in Pseudomonas syringae.

The paper examines how host-type heterogeneity and immune-mediated competition affect pathogen diversity using a theoretical model inspired by rotavirus.

The paper updates an epidemiological model to predict influenza incidence for the 2018-2019 season and evaluates its accuracy for the 2017-2018 season.

The paper demonstrates a method to forecast seasonal H3N2 influenza incidence by integrating evolutionary changes with an epidemiological model, achieving accurate real-time predictions for the 2016/2017 season.

The paper uses a stochastic model to study how competition and reassortment affect rotavirus strain structure, finding that new strains displace existing ones in open antigenic niches.

The paper identifies single amino-acid polymorphisms in PfEMP1 homology blocks associated with severe malaria pathophysiology and disease outcomes.