Kanchan Garai

This study shows apoE4 accelerates tau condensate to amyloid transition, while lipidation reduces its effects, suggesting lipidation enhancement could counteract its pathogenic role.

This study uses single molecule microscopy to show that tau condensate ageing involves liquid-solid coexistence at the fibril level, with spatial heterogeneity driving the transition.

This paper shows that Alzheimer's amyloid-beta peptide forms biomolecular condensates on lipid bilayers, accelerating amyloid aggregation and revealing a new early step in the disease mechanism.