Author

Stephen D’Amico

Stony Brook MedicineORCID 0009-0004-6425-7822Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Approach:statistical model· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

Nancy C. ReichiD· 2 shared Varvara KirilloviD· 2 shared Brian S. SheridaniD· 1 shared Hong QiniD· 1 shared Jingxuan LiuiD· 1 shared Oleksi PetrenkoiD· 1 shared Xinyuan LeiiD· 1 shared Zhijuan QiuiD· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

8 top collaborators · 23 mutual co-author links

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2 shared papersCancer Biology

Nancy C. ReichORCID

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2 papers

Recent·Most cited

Cancer BiologyEMBO ReportsPeer-reviewedAug 26, 2025

STAT3 Sustains Tumorigenicity Following Mutant KRAS Ablation

Stephen D’Amico, Varvara Kirillov, Jingxuan Liu…·Nancy C. Reich

This study shows that STAT3 sustains tumorigenicity in KRAS-ablated pancreatic cancer cells, highlighting its role in maintaining malignant identity and suggesting it as a therapeutic target.

2 citations

mousebulk RNA-seq

𝕏 PostBluesky

Cancer BiologyeLifePeer-reviewedJan 30, 2024

STAT3 is a genetic modifier of TGF-beta induced EMT in KRAS mutant pancreatic cancer

Stephen D’Amico, Varvara Kirillov, Oleksi Petrenko, Nancy C. Reich

The paper identifies STAT3 as a genetic modifier of TGF-beta-induced EMT in KRAS mutant pancreatic cancer, showing its role in tumor maintenance and epithelial homeostasis.

11 citations

humanbulk RNA-seq

𝕏 PostBluesky