Author

Ken Matsuoka

The University of OsakaORCID 0009-0006-9485-7188Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Bioinformatics· 1 Cell Biology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Assay:ATAC-seq· 2 Research Focus:differential expression· 2 Organism:human· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Approach:deep learning· 1 Approach:experimental assay· 1

Frequent Co-Authors

Hijiri Inoue· 3 shared Hisakazu KatoiD· 3 shared Seiji TakashimaiD· 3 shared Chisato OkamotoiD· 2 shared Osamu TsukamotoiD· 2 shared Akira OhtakeiD· 1 shared Ann E. FrazieriD· 1 shared Atsuhito TakedaiD· 1 shared

Activity by Year

2025-2025 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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3 shared papersMolecular Biology

Hijiri Inoue

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3 papers

Recent·Most cited

Molecular BiologybioRxivPreprintOct 13, 2025

A Conserved Stress-Inducible Hub Enhancer Governs Natriuretic Peptide Expression and Undergoes Dynamic and Reversible Activation in Human Heart Failure

Ken Matsuoka, Hijiri Inoue, Jo Hyejin…·Osamu Tsukamoto

This study identifies CR9 as a stress-inducible hub enhancer critical for natriuretic peptide expression in heart failure, showing its reversible activation in human hearts.

DOI 10.1101/2025.10.13.682224

humanChIP-seq

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BioinformaticsbioRxivPreprintOct 11, 2025

HiFAA: a high-confidence framework for transcription factor footprinting with ATAC-seq

Hijiri Inoue, Chisato Okamoto, Hisakazu Kato…·Ken Matsuoka

HiFAA improves TF footprint detection from ATAC-seq by integrating domain-restricted analysis, single-base resolution, and footprint ranking, enhancing sensitivity and specificity.

DOI 10.1101/2025.10.11.681777

ATAC-seqstatistical model

𝕏 PostBluesky

Cell BiologyScience AdvancesPeer-reviewedApr 4, 2025

Atf3 controls transitioning in female mitochondrial cardiomyopathy as identified by single-cell transcriptomics

Tasneem Qaqorh, Yusuke Takahashi, Kohei Sameshima…·Yasunori Shintani

This study identifies Atf3 as a key regulator of transition from compensation to dysfunction in female mitochondrial cardiomyopathy using single-cell transcriptomics.

9 citations

mousehuman

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