Author

Jovana Rajak

University Hospital UlmORCID 0009-0007-0229-8583Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

Charlotte Marie NiemeyeriD· 2 shared Christian FlothoiD· 2 shared Daniel B. LipkaiD· 2 shared Miriam ErlacheriD· 2 shared Sheila BohleriD· 2 shared Abdul‐Habib MaagiD· 1 shared Alexandra Emilia SchlaakiD· 1 shared Anna Lena Stippel· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersCancer Biology

Charlotte Marie NiemeyerORCID

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2 papers

Recent·Most cited

Cancer BiologyBlood Cancer DiscoveryPeer-reviewedDec 5, 2025

Oncogenic RAS-Pathway Activation Drives Oncofetal Reprogramming and Creates Therapeutic Vulnerabilities in Juvenile Myelomonocytic Leukemia

Mark Hartmann, Maximilian Schönung, Jovana Rajak…·Daniel B. Lipka

This study identifies oncofetal reprogramming in juvenile myelomonocytic leukemia, linking RAS-pathway activation to fetal gene expression and revealing CD52 as a therapeutic target.

9 citationstop 5%

humanbulk RNA-seq

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Cancer BiologybioRxivPreprintOct 1, 2025

Oncogenic PTPN11/SHP2 drives immune escape in juvenile myelomonocytic leukemia (JMML) through activation of ectonucleotidase/adenosine signaling

Jovana Rajak, Ke Meng, Anna Lena Stippel…·Miriam Erlacher

This study identifies PTPN11/SHP2-driven adenosine signaling as a mechanism of immune escape in JMML, showing that CD39 inhibition can enhance T cell responses and reduce myeloproliferation.

DOI 10.1101/2025.10.01.679664

mousebulk RNA-seq

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