Author

A. Anjum

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Research Focus:metabolism & bioenergetics· 1 Research Focus:protein folding & dynamics· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Leunig· 1 shared A. Titova· 1 shared B. Dhayalan· 1 shared D. A. Ostrov· 1 shared D. Larkin· 1 shared D. Rossaro· 1 shared E. Riedlinger· 1 shared J. Sun· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperImmunology

A. Leunig

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Thrombocytopenia and splenic platelet directed immune responses after intravenous ChAdOx1 nCov-19 administration

L. Nicolai, A. Leunig, K. Pekayvaz, A. Anjum, E. Riedlinger, L. Eivers, M.-L. Hoffknecht, D. Rossaro, R. Escaig, R. Kaiser, V. Polewka, A. Titova, K. Spiekermann, M. Iannacone, K. Stark, S. Massberg

This study investigates how intravenous ChAdOx1 nCov-19 vaccination leads to thrombocytopenia and immune responses against platelets, suggesting intramuscular injection as a safer alternative.

bioRxiv · Jun 29, 2021Immunology

Role of Proinsulin Self-Association in Mutant INS gene-induced Diabetes of Youth

M. Liu, P. Arvan, J. Sun, Y. Xiong, X. Li, L. Haataja, W. Chen, S. A. Mir, R. Madley, D. Larkin, A. Anjum, B. Dhayalan, N. K. Rege, N. D. Wickramasinghe, M. A. Weiss, , D. A. Ostrov

This study identifies proinsulin self-association as a mechanism driving dominant-negative effects of misfolded proinsulin in MIDY, offering a therapeutic target for enhancing insulin production.

bioRxiv · Oct 1, 2019Cell Biology