Author

A. Dickmanns

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cell Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:bacteria (other)· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:development & morphogenesis· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1

Frequent Co-Authors

R. Ficner· 2 shared A. Chari· 1 shared A. Filosa· 1 shared A. R. Pearson· 1 shared B. Fahrenkrog· 1 shared B. Vanhollebeke· 1 shared C. Berndt· 1 shared C. Herzberg· 1 shared

Activity by Year

2018-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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Curved chords = mutual co-authorship

2 shared papersMicrobiology

R. Ficner

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Redox regulation of the SARS-CoV-2 main protease provides new opportunities for drug design

K. Tittmann, L.-M. Funk, G. Poschmann, A. Chari, F. Rabe von Pappenheim, K. M. Stegmann, A. Dickmanns, N. Eulig, M. Wensien, E. Paknia, G. Heyne, E. Penka, A. R. Pearson, C. Berndt, T. Fritz, , U. Curth

This study identifies redox regulation of SARS-CoV-2 Mpro and discovers antiviral reagents that mimic redox switching, suggesting new drug targets.

bioRxiv · Apr 18, 2022 · PreprintBiochemistry

A rendezvous of two second messengers: The c-di-AMP receptor protein DarB controls (p)ppGpp synthesis in Bacillus subtilis

L. Krueger, C. Herzberg, D. Wicke, H. Baehre, J. L. Heidemann, A. Dickmanns, K. Schmitt, R. Ficner, J. Stuelke

This study identifies DarB as a c-di-AMP receptor that interacts with Rel to regulate (p)ppGpp synthesis in Bacillus subtilis under low potassium conditions.

bioRxiv · Aug 27, 2020Microbiology

Mutations in nucleoporin NUP88 cause lethal neuromuscular disorder

E. Bonnin, P. Cabochette, A. Filosa, R. Juehlen, S. Komatsuzaki, M. Hezwani, A. Dickmanns, V. Martinelli, M. Vermeersch, L. Supply, N. Martins, L. Pirenne, G. Ravenscroft, M. Lombard, S. A. Port, , B. Fahrenkrog

This study identifies biallelic NUP88 mutations as a cause of lethal fetal akinesia deformation sequence (FADS) and shows their effects on neuromuscular development in zebrafish and human cells.

bioRxiv · Aug 14, 2018Cell Biology