Author

A. FROBERT

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1

Frequent Co-Authors

G. AJALBERT· 2 shared M.-N. Giraud· 2 shared S. Cook· 2 shared A. Brenna· 1 shared B. Fellay· 1 shared C. KALTENRIEDER· 1 shared D. M. Potenza· 1 shared I. Borrego· 1 shared

Activity by Year

2022-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersPhysiology

G. AJALBERT

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Cell-autonomous and non-cell-autonomous effects of arginase-II on cardiac aging

D. M. Potenza, X. Cheng, G. AJALBERT, A. Brenna, M.-N. Giraud, A. FROBERT, S. Cook, K. D. Mertz, Z. Yang, X.-F. Ming

This study identifies non-cell-autonomous and cell-autonomous roles of arginase-II in cardiac aging, showing its impact on inflammation, fibrosis, and ischemic injury through macrophage-derived IL-1β and fibroblast mtROS.

bioRxiv · Jan 17, 2024 · PreprintPhysiology

Fibrin, Bone Marrow Cells and macrophages interactively modulate cardiomyoblast fate.

I. Borrego, A. FROBERT, G. AJALBERT, J. VALENTIN, C. KALTENRIEDER, B. Fellay, M. STUMPE, S. Cook, J. DENGJEL, M.-N. GIRAUD

This study shows that fibrin and bone marrow cells reduce cardiac damage and improve function after myocardial infarction by modulating macrophage behavior and promoting cardiomyoblast proliferation.

bioRxiv · Jan 6, 2022Physiology