Author

A. Gutwillig

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Research Focus:cell type annotation· 3 Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Approach:deep learning· 1 Research Focus:drug response / drug discovery· 1 Assay:flow cytometry· 1

Frequent Co-Authors

D. Rasoulouniriana· 2 shared L. Farhat-Younis· 2 shared N. Santana-Magal· 2 shared P. Rider· 2 shared Y. Carmi· 2 shared A. Ashkenazi· 1 shared A. Bart· 1 shared A. Barzel· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersImmunology

D. Rasoulouniriana

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Expression of modified FcγRI enables myeloid cells to elicit robust tumor-specific cytotoxicity

Y. Carmi, L. Farhat-Younis, M. Na, A. Zarfin, N. Santana-Magal, A. Richter, A. Khateeb, A. Gutwillig, D. Rasoulouniriana, A. Gleiberman, L. Beck, T. Giger, A. Ashkenazi, A. Barzel, P. Rider

This study modifies myeloid cells to elicit tumor-specific cytotoxicity using chimeric receptors that signal through IgM and bind tumor antigens.

bioRxiv · Sep 30, 2023Immunology

A novel deep learning pipeline for cell typing and phenotypic marker quantification in multiplex imaging

E. Markovits, T. Dankovich, R. Gluskin, I. Weiss, A. Gutwillig, T. Dicker, S. Dagan, R. Elran, B. Arbiv, Y. Shachaf, A. Bart, A. Debby, N. Asher, G. Ben-Betzalel, R. Shapira-Frommer, , O. Zelichov

This paper introduces a deep learning pipeline for cell typing and phenotypic marker quantification in multiplex imaging, achieving high accuracy across different tumor types and imaging modalities.

bioRxiv · Nov 9, 2022 · PreprintImmunology

Transient cell-in-cell formation underlies tumor resistance to immunotherapy

A. Gutwillig, N. Santana-Magal, L. Farhat-Younis, D. Rasoulouniriana, A. Madi, C. Luxenburg, J. Cohen, K. Padmanabhan, N. Shomron, G. Shapira, M. Feinmesser, O. Zlotnik, R. Debets, N. E. Reticker-Flynn, P. Rider, Y. Carmi

This study identifies transient cell-in-cell formations as a mechanism enabling tumor resistance to immunotherapy, showing they protect tumor cells from immune and chemotherapy attacks.

bioRxiv · Sep 10, 2020 · PreprintImmunology