Author

A. Kuntze

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1

Frequent Co-Authors

A. Schlichting· 1 shared A. Schwab· 1 shared A. Stevens· 1 shared B. Altvater· 1 shared B. Fels· 1 shared C. Roessig· 1 shared C. Scholl· 1 shared D. Ing· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperPhysiology

A. Schlichting

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Exploiting WEE1 kinase activity as FUS::DDIT3-dependent therapeutic vulnerability in myxoid liposarcoma

L. Heinst, K. S. Lee, R. Berthold, I. Isfort, S. Wosnig, A. Kuntze, S. Hafner, B. Altvater, C. Roessig, P. Aman, E. Wardelmann, C. Scholl, W. Hartmann, S. Froehling, M. Trautmann

This study identifies WEE1 kinase activity as a therapeutic target in FUS::DDIT3-driven myxoid liposarcoma by showing its role in replication stress tolerance and cell survival.

Clinical Cancer Research · Aug 29, 2024Peer-reviewedCancer Biology

Piezo1-induced durotaxis of pancreatic stellate cells depends on TRPC1 and TRPV4 channels

I. Budde, A. Schlichting, D. Ing, S. Schimmelpfennig, A. Kuntze, B. Fels, J. M.-J. Romac, S. M. Swain, R. A. Liddle, A. Stevens, A. Schwab, Z. Petho

This study identifies Piezo1, TRPC1, and TRPV4 as mechanosensitive ion channels crucial for durotaxis in pancreatic stellate cells, which contributes to pancreatic cancer progression.

bioRxiv · Dec 22, 2023Physiology