Author

A. M. Fry

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Assay:proteomics· 3 Research Focus:cell signaling & trafficking· 2 Organism:human· 2 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

K. R. Straatman· 2 shared L. O'Regan· 2 shared M. Richards· 2 shared R. Adib· 2 shared R. Bayliss· 2 shared A. A. Holder· 1 shared A. K. Subodhi· 1 shared A. Pain· 1 shared

Activity by Year

2018-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersCell Biology

K. R. Straatman

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Plasmodium NEK1 coordinates MTOC organisation and kinetochore attachment during rapid mitosis in male gamete formation

M. Zeeshan, R. Rashpa, D. J. Ferguson, G. Mckeown, R. Nugmanova, A. K. Subodhi, R. Beyeler, S. L. Pashley, R. Markus, D. Brady, M. Roques, A. R. Bottrill, A. M. Fry, A. Pain, s. vaughan, , R. Tewari

This study identifies NEK1's role in organizing the MTOC and ensuring kinetochore attachment during rapid mitosis in Plasmodium male gamete formation.

bioRxiv · Feb 5, 2024 · PreprintCell Biology

Endogenous oncogenic KRAS expression increases cell proliferation and motility in near-diploid hTERT RPE-1 cells

N. L. Hindul, L. R. Abbott, S. M. D. Adan, K. R. Straatman, A. M. Fry, K. Hirota, K. Tanaka

This study shows that endogenous oncogenic KRAS mutations increase cell proliferation and motility in hTERT RPE-1 cells, with differential responses to targeted therapies.

bioRxiv · Sep 8, 2023Cell Biology

EML4-ALK V3 drives cell migration through NEK9 and NEK7 kinases in non-small-cell lung cancer

L. O'Regan, G. Barone, R. Adib, C. G. Woo, H. J. Jeong, E. Richardson, M. Richards, P. Muller, S. Collis, D. Fennell, J. Choi, R. Bayliss, A. M. Fry

The paper shows EML4-ALK V3 promotes cell migration via NEK9 and NEK7 kinases, leading to worse outcomes in lung cancer.

bioRxiv · Mar 4, 2019 · PreprintCancer Biology

Mitotic phosphorylation by Nek6 and Nek7 reduces microtubule affinity of EML4 to alter spindle dynamics and promote chromosome congression

A. M. Fry, R. Adib, J. M. Montgomery, J. Atherton, L. O'Regan, M. Richards, K. R. Straatman, D. Roth, A. Straube, R. Bayliss, C. A. Moores

bioRxiv · Nov 9, 2018Cell Biology