Author

A. M. Rivera

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Microbiology· 3

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:ecology & biodiversity· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

C. M. Theriot· 3 shared J. R. Fletcher· 2 shared R. J. Parsons· 2 shared S. A. Montgomery· 2 shared A. D. Patterson· 1 shared A. D. Reed· 1 shared A. Shen· 1 shared A. Stewart· 1 shared

Activity by Year

2020-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersMicrobiology

C. M. Theriot

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

The Stickland fermentation precursor trans-4-hydroxyproline differentially impacts the metabolism of Clostridioides difficile and commensal Clostridia

C. M. Theriot, A. D. Reed, J. R. Fletcher, Y. Huang, R. Thanissery, A. M. Rivera, R. J. Parsons, A. Stewart, D. J. Kountz, A. Shen, E. P. Balskus

This study investigates how trans-4-hydroxyproline affects the metabolism of C. difficile and commensal Clostridia, revealing differential gene expression and microbiota changes in mice.

bioRxiv · Nov 12, 2021 · PreprintMicrobiology

Clostridioides difficile exploits toxin-mediated inflammation to alter the host nutritional landscape and exclude competitors from the gut microbiota

J. R. Fletcher, C. M. Pike, R. J. Parsons, A. M. Rivera, M. H. Foley, M. R. McLaren, S. A. Montgomery, C. M. Theriot

This study shows C. difficile uses toxin-induced inflammation to alter host nutrient availability and outcompete Bacteroides by utilizing collagen-derived amino acids.

bioRxiv · Aug 6, 2020Microbiology

Ursodeoxycholic acid (UDCA) mitigates the host inflammatory response during Clostridioides difficile infection by altering gut bile acids which attenuates NF-κB signaling via bile acid activated receptors

J. A. Winston, A. M. Rivera, J. Cai, R. M. Thanissery, S. A. Montgomery, A. D. Patterson, C. M. Theriot

This paper shows that UDCA reduces inflammation during CDI by altering bile acids and modulating NF-κB signaling through FXR and TGR5 receptors.

Infection and Immunity · May 20, 2020Peer-reviewedMicrobiology