Author

A. Markazi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Assay:metabolomics· 1 Assay:metagenomics· 1 Assay:proteomics· 1 Organism:rat· 1

Frequent Co-Authors

S.-J. Gao· 2 shared J. Feng· 1 shared L. Chen· 1 shared M. S. McGrath· 1 shared P. Bracci· 1 shared W. Meng· 1 shared W. Yuan· 1 shared X. ZhangiD· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 24 mutual co-author links

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2 shared papersMicrobiology

S.-J. Gao

Mutual co-authors in cluster

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2 papers

Recent·Most cited

N6-methyladenosine (m6A) and reader protein YTHDF2 enhance innate immune response by mediating DUSP1 mRNA degradation and activating mitogen-activated protein kinases during bacterial and viral infections

S.-J. Gao, J. Feng, W. Meng, L. Chen, X. Zhang, A. Markazi, W. Yuan, Y. Huang

The paper shows that m6A and YTHDF2 regulate DUSP1 to enhance innate immune response during infections by promoting DUSP1 degradation and activating MAPKs.

mBio · Feb 28, 2023Peer-reviewedMicrobiology

Pseudomonas aeruginosa Stimulates Inflammation and Enhances KSHV-Induced Cell Proliferation and Cellular Transformation through Both LPS and Flagellin

A. Markazi, P. Bracci, M. S. McGrath, S.-J. Gao

This study shows that Pseudomonas aeruginosa enhances inflammation and cell proliferation in KSHV-transformed cells via LPS and flagellin, highlighting potential therapeutic targets for AIDS-KS patients.

bioRxiv · Oct 7, 2020Cancer Biology