Author

A. Metais

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 3 Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:bacteria (other)· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:development & morphogenesis· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

A. Blondel· 1 shared A. Boyance· 1 shared A. C. Koong· 1 shared A. Coste· 1 shared A. Diz-Munoz· 1 shared A. Hessel· 1 shared A. Papaioannou· 1 shared C. Berrone· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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Curved chords = mutual co-authorship

1 shared paperBiochemistry

A. Blondel

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

The membrane-actin linkers ezrin, radixin, and moesin are dispensable for macrophage migration and cortex mechanics.

P. Verdys, J. Rey Barroso, J. Vermeil, M. Bergert, T. Sanchez, A. Metais, T. Mangeat, E. Bellard, C. Bigot, J.-P. Girard, I. Maridonneau-Parini, C. Verollet, F. Lagarrigue, A. Diz-Munoz, J. Heuvingh, , R. Poincloux

This study shows that ezrin, radixin, and moesin are not required for macrophage migration or actin cortex mechanics, challenging their universal importance in these processes.

bioRxiv · Jul 27, 2023 · PreprintCell Biology

Phospholipid peroxidation fuels ExoU phospholipase-dependent cell necrosis and supports Pseudomonas aeruginosa-driven pathology

S. Bagayoko, S.-A. Leon-Icaza, M. Pinilla, A. Hessel, K. Santoni, P.-J. Bordignon, F. Moreau, E. Eren, A. Boyance, E. Naser, L. Lefevre, C. Berrone, N. Iacobachvili, A. Metais, Y. Rombouts, , E. Meunier

This study identifies phospholipid peroxidation as a mechanism enhancing ExoU phospholipase activity, leading to cell necrosis and supporting Pseudomonas aeruginosa pathogenesis.

bioRxiv · Feb 17, 2021Immunology

Stress-induced tyrosine phosphorylation of RtcB modulates IRE1 activity and signaling outputs.

A. Papaioannou, F. G. Centonze, A. Metais, M. Maurel, L. Negroni, M. Gonzales-Quiroz, E. Zare Golchesmeh, A. Blondel, A. C. Koong, C. Hetz, R. Pedeux, M. L. Tremblay, S. J. Mahdizadeh, G. Svensson, L. A. Eriksson, e. chevet

This study identifies RtcB tyrosine phosphorylation as a regulatory mechanism controlling IRE1's dual RNase activities, influencing cell survival or death during ER stress.

bioRxiv · Mar 2, 2020Biochemistry