Author

A. Petry

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1 Cancer Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Research Focus:host-pathogen & immune response· 1 Assay:metagenomics· 1

Frequent Co-Authors

A. C. Ericsson· 1 shared A. Goerlach· 1 shared A. L. Russell· 1 shared A. Sipol· 1 shared B. B. Kragelund· 1 shared B. Xue· 1 shared C. Prexler· 1 shared D. Brenner· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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1 shared paperMicrobiology

A. C. Ericsson

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Supplier-origin gut microbiomes affect host body weight and select autism-related behaviors

Z. L. McAdams, K. Gustafson, A. L. Russell, R. Self, A. Petry, T. Lever, A. C. Ericsson

The paper shows that gut microbiome diversity from different suppliers affects body weight and autism-related behaviors in BTBR mice, with a male-specific behavioral impact.

bioRxiv · Apr 1, 2024 · PreprintMicrobiology

The prolactin receptor scaffolds Janus kinase 2 via co-structure formation with phosphoinositide-4,5-bisphosphate

R. Araya-Secchi, K. Bugge, P. Seiffert, A. Petry, G. W. Haxholm, K. Lindorff-Larsen, S. F. Pedersen, L. Arleth, B. B. Kragelund

This study shows that the prolactin receptor forms a co-structure with JAK2 and PI(4,5)P2, enabling signal transduction by stabilizing the receptor's disordered domain.

bioRxiv · Nov 23, 2022Biophysics

MondoA Drives Malignancy in cALL through Enhanced Adaptation to Metabolic Stress

A. Sipol, E. Hameister, B. Xue, A. Petry, C. Prexler, M. Barenboim, R. A. Rubio, M. C. Baldauf, D. G. Franchina, J. Schmaeh, G. Richter, G. Cario, T. G. P. Gruenewald, A. Goerlach, D. Brenner, , S. E. G. Burdach

This study shows MondoA promotes malignancy in pediatric cALL by enhancing metabolic stress adaptation, with potential therapeutic implications.

bioRxiv · Nov 1, 2020Cancer Biology