Author

A. Philips

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell type annotation· 1 Organism:Drosophila· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1 Assay:long-read sequencing· 1

Frequent Co-Authors

A. A. Rahim· 1 shared A. Dowsey· 1 shared A. E. Burrows· 1 shared A. Sharaireh· 1 shared A. Tierney· 1 shared C. D. Thornton· 1 shared D. Oliver· 1 shared D. Portwood· 1 shared

Activity by Year

2021-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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1 shared paperCell Biology

A. A. Rahim

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

CLN7 mutation causes aberrant redistribution of protein isoforms and contributes to Batten disease pathobiology

A. Sharaireh, M. Guevara Ferrer, S. Herranz-Martin, M. Garcia-Macia, A. Philips, A. Tierney, M. P. Hughes, O. Coombe-Tennant, H. Nelvagel, A. E. Burrows, S. Fielding, L. M. FitzPatrick, C. D. Thornton, S. Storch, S. E. Mole, , T. R. McKay

This study shows CLN7 mutations cause nuclear and endolysosomal protein distribution defects in Batten disease, leading to apoptosis and neuroinflammation, suggesting combinatorial therapies may be needed.

bioRxiv · Apr 21, 2022 · PreprintCell Biology

Chemically modified dsRNA induces RNAi effects in insects in vitro and in vivo: A potential new tool for improving RNA-based plant protection

J. D. Howard, M. Beghyn, N. Dewulf, Y. De Vos, A. Philips, D. Portwood, P. M. Kilby, D. Oliver, W. Maddelein, S. Brown, M. J. Dickman

This study shows chemically modified long dsRNA improves RNAi efficacy in insects, leading to mortality in both cell cultures and live insects.

bioRxiv · Feb 2, 2022Bioengineering

Profile of basal cell carcinoma mutations and copy number alterations - focus on gene-associated noncoding variants

P. M. Nawrocka, P. Galka-Marciniak, M. O. Urbanek-Trzeciak, I. M., N. Szostak, A. Philips, L. Susok, M. Sand, P. Kozlowski

This study analyzes coding and noncoding mutations in basal cell carcinoma, finding that noncoding variants account for ~50% of mutations and identify new cancer drivers.

bioRxiv · Jun 24, 2021Cancer Biology