Author

B. Button

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell-cell communication· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Organism:human· 1

Frequent Co-Authors

C. B. Morrison· 2 shared C. Ehre· 2 shared D. B. Hill· 2 shared M. R. Markovetz· 2 shared B. R. Grubb· 1 shared G. Radicioni· 1 shared G. V. Stoychev· 1 shared H. P. Goodell· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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2 shared papersPathology

C. B. Morrison

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Hyperconcentrated Mucus Unifies Submucosal Gland and Superficial Airway Dysfunction in Cystic Fibrosis

T. Kato, G. Radicioni, M. J. Papanikolas, G. V. Stoychev, M. R. Markovetz, K. Aoki, M. Porterfield, K. Okuda, S. M. B. Cardenas, R. C. Gilmore, C. B. Morrison, C. Ehre, K. A. Burns, K. K. White, T. A. Brennan, , R. C. Boucher

This study identifies hyperconcentrated mucus as a unifying mechanism for submucosal gland and superficial airway dysfunction in cystic fibrosis.

bioRxiv · Sep 22, 2021 · PreprintPathology

BPIFB1 loss alters airway mucus properties and diminishes mucociliary clearance.

L. J. Donoghue, M. R. Markovetz, C. B. Morrison, K. M. McFadden, T. D. Rogers, B. Button, C. Ehre, D. B. Hill, B. R. Grubb, S. N. P. Kelada

This study shows that BPIFB1 loss in mice impairs mucociliary clearance by altering mucus viscosity without affecting ion transport or ciliary beat frequency.

bioRxiv · Jun 17, 2020Physiology