Author

B. Petrova

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 1 Cell Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:drug response / drug discovery· 2 Approach:experimental assay· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

A. Cameron· 1 shared A. Lawless· 1 shared A. M. Cicerchia· 1 shared A. Mehta· 1 shared A. Tal-Mason· 1 shared B. M. Markus· 1 shared C. Harding· 1 shared C. Palin· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCell Biology

A. Cameron

Author profile →

3 papers

Recent·Most cited

Disrupting CD38-driven T cell dysfunction restores sensitivity to cancer immunotherapy

O.-Y. Revach, A. M. Cicerchia, O. Shorer, B. Petrova, S. Anderson, J. Park, L. Chen, A. Mehta, S. J. Wright, N. McNamee, A. Tal-Mason, G. Cattaneo, P. Tiwari, H. Xie, J. M. Sweere, , K. Yizhak

This study shows that targeting CD38 improves T cell function and restores response to immunotherapy in melanoma models.

bioRxiv · Feb 12, 2024 · PreprintCancer Biology

FDX1 regulates cellular protein lipoylation through direct binding to LIAS.

M. Dreishpoon, N. Bick, B. Petrova, D. M. Warui, A. Cameron, S. J. Booker, N. Kanarek, T. Golub, P. Tsvetkov

This study identifies FDX1 as a direct regulator of protein lipoylation by binding to LIAS, essential for cell survival under low glucose conditions.

bioRxiv · Feb 3, 2023Cell Biology

Genetic screens reveal a central role for heme biosynthesis in artemisinin susceptibility

C. Harding, S. M. Sidik, B. Petrova, N. F. Gnädig, J. Okombo, K. E. Ward, B. M. Markus, D. A. Fidock, S. Lourido

The paper uses CRISPR screens to identify genetic factors affecting artemisinin susceptibility in apicomplexan parasites, revealing heme biosynthesis as a key pathway.

bioRxiv · Aug 28, 2019Microbiology