Author

B.-H. Lee

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Microbiology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Approach:biophysical modeling· 1 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

D. Rigaudeau· 2 shared E. Duchaud· 2 shared J.-F. Bernardet· 2 shared T. Rochat· 2 shared B. Fradet· 1 shared D. Finley· 1 shared D. Van Dalen· 1 shared E. Quillet· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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2 shared papersPathology

D. Rigaudeau

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Interplay between a bacterial pathogen and its host in rainbow trout isogenic lines with contrasted susceptibility to Cold Water Disease

B.-H. Lee, E. Quillet, D. Rigaudeau, N. Dechamp, E. Duchaud, J.-F. Bernardet, P. Boudinot, T. Rochat

The paper compares BCWD resistance in rainbow trout isogenic lines, finding that resistant fish have lower bacterial growth rates and higher expression of immune genes like arg2 and tlr2/tlr7.

bioRxiv · Feb 23, 2023Pathology

Investigation of the genus Flavobacterium as a reservoir for fish-pathogenic bacterial species: the case of Flavobacterium collinsii

E. Duchaud, B.-H. Lee, P. Nicolas, I. B. Saticioglu, B. Fradet, J.-F. Bernardet, D. Rigaudeau, T. Rochat

This study identifies and characterizes Flavobacterium collinsii as a potential opportunistic fish pathogen, showing it can colonize stressed fish but not cause clinical disease under normal conditions.

bioRxiv · Sep 27, 2022 · PreprintMicrobiology

Allosteric control of Ubp6 and the proteasome via a bidirectional switch

K. Y. S. Hung, S. Klumpe, M. R. Eisele, S. Elsasser, G. Tian, S. Sun, J. A. Moroco, T. C. Cheng, T. Joshi, T. Seibel, D. Van Dalen, X.-H. Feng, Y. Lu, H. Ovaa, J. R. Engen, , D. Finley

This study identifies an allosteric switch that controls Ubp6 and the proteasome, showing their active states are mutually exclusive.

bioRxiv · Jul 16, 2021Biochemistry