Author

C. M. Cuda

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 3

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Research Focus:differential expression· 3 Approach:experimental assay· 3 Approach:statistical model· 3 Research Focus:cell-cell communication· 2 Organism:human· 2 Assay:scRNA-seq· 2 Assay:bulk RNA-seq· 1

Frequent Co-Authors

H. Perlman· 2 shared A. B. Montgomery· 1 shared A. Bharat· 1 shared A. C. McQuattie-Pimentel· 1 shared A. Misharin· 1 shared A. R. Burg· 1 shared A. R. Shields· 1 shared A. S. Flozak· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 15 mutual co-author links

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2 shared papersImmunology

H. Perlman

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Single cell transcriptomic analysis of renal allograft rejection reveals novel insights into intragraft TCR clonality

T. Shi, A. R. Burg, J. T. Caldwell, K. Roskin, C. M. C. Rojas, P. C. Chukwuma, G. I. Gray, S. G. Foote, J. Alonso, C. M. Cuda, D. A. Allman, J. S. Rush, C. H. Regnier, G. Wieczorek, R. R. Alloway, , D. A. Hildeman

The paper uses single-cell RNA sequencing and TCR sequencing to analyze renal allograft rejection, finding CD8+ T cell clonal expansion linked to rejection, independent of immunosuppression type.

bioRxiv · Feb 8, 2023Immunology

Tissue-resident, extravascular CD64-Ly6C- population forms a critical barrier for inflammation in the synovium

A. B. Montgomery, S.-Y. Chen, G. Gadhvi, M. G. Mayr, C. M. Cuda, S. Dominguez, H. M. Makinde, A. Misharin, M. G. Gurra, M. AKBARPOUR, A. Bharat, G. R. S. Bundinger, D. R. Winter, H. Perlman

The paper identifies and characterizes two distinct synovial monocyte populations, showing that the extravascular population is critical for RA-like disease development.

bioRxiv · Feb 25, 2021 · PreprintImmunology

Single-cell RNA-seq reveals spatially restricted multicellular fibrotic niches during lung fibrosis

N. Joshi, S. Watanabe, R. Verma, R. P. Jablonski, C.-I. Chen, P. Cheresh, P. A. Reyfman, A. C. McQuattie-Pimentel, L. Sichizya, A. S. Flozak, C. J. Gottardi, C. M. Cuda, H. Perlman, M. Jain, D. W. Kamp, , A. V. Misharin

The paper identifies monocyte-derived alveolar macrophages as causally linked to fibrosis in lung tissue through autocrine signaling and fibroblast activation.

European Respiratory Journal · Jan 1, 2020Peer-reviewedImmunology