Author

L. Luzi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:scRNA-seq· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Organism:human· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

P. G. Pelicci· 2 shared A. Cossa· 1 shared A. Dalmasso· 1 shared A. Polazzi· 1 shared A. Tirelli· 1 shared C. Priami· 1 shared D. Bossi· 1 shared E. Gatti· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersCancer Biology

P. G. Pelicci

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2 papers

Recent·Most cited

IN VIVO ISOLATION OF A QUIESCENT MELANOMA POPULATION WITH INVASIVE PROPERTIES UNVEILS A TRANSCRIPTIONAL REPROGRAMMING DRIVEN BY THE TUMOR NICHE

L. Lanfrancone, F. Lotti, M. Meliksetyan, M. Malferrari, N. Quaresima, S. Rapino, V. Mollo, I. Ferrarotto, T. Vlachou, D. Bossi, P. G. Pelicci, L. Luzi

The paper isolates a quiescent melanoma population in vivo, showing it has invasive properties and undergoes transcriptional reprogramming driven by the tumor niche.

bioRxiv · Jul 31, 2023 · PreprintCancer Biology

The breast cancer pro-metastatic phenotype requires concomitant hyper-activation of ECM remodeling and dsRNA-IFN1 signaling in rare clone cells

N. Roda, A. Cossa, R. Hillje, A. Tirelli, F. Ruscitto, S. Cheloni, C. Priami, A. Dalmasso, V. Gambino, G. Blandano, A. Polazzi, P. Falvo, E. Gatti, L. Mazzarella, L. Luzi, , P. G. Pelicci

This study identifies that the breast cancer pro-metastatic phenotype arises from rare clones with hyper-activation of ECM remodeling and dsRNA-IFN1 signaling, and shows these genes predict metastasis progression.

bioRxiv · Jul 13, 2022Cancer Biology