Author

C. Regnault

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:drug response / drug discovery· 1 Assay:metabolomics· 1 Organism:plant (other)· 1

Frequent Co-Authors

A. Abraham· 1 shared A. Kar· 1 shared A. Korula· 1 shared A. P. Waters· 1 shared A. R. Barr· 1 shared A. T. Saurin· 1 shared A. Venkatraman· 1 shared A. W. Guiguemde· 1 shared

Activity by Year

2022-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 12 mutual co-author links

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Curved chords = mutual co-authorship

1 shared paperCancer Biology

A. Abraham

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Targeting Metabolic Dependencies in Acute Myeloid Leukemia: A Dual Strategy using Arsenic Trioxide and Artesunate.

N. Balasundaram, A. Venkatraman, Y. Augustin, H. K. Palani, C. Regnault, A. Korula, U. P. Kulkarni, E. S. Edison, P. Balasubramanian, B. George, A. Abraham, S. Krishna, V. Mathews

This study investigates the combination of artesunate and arsenic trioxide to treat AML with reduced off-target effects on normal hematopoietic stem cells.

bioRxiv · Jun 23, 2024 · PreprintCancer Biology

A conserved metabolic signature associated with response to fast-acting antimalarial agents

N. V. Simwela, A. W. Guiguemde, J. Straimer, C. Regnault, F. Yokokawa, B. Taft, T. T. Diagana, M. P. Barrett, A. P. Waters

This paper identifies a conserved metabolic signature in malaria parasites exposed to fast-acting antimalarial drugs, suggesting a common mechanism of action.

bioRxiv · Oct 3, 2022 · PreprintMicrobiology

Cell overgrowth during G1 arrest triggers an osmotic stress response and chronic p38 activation to promote cell cycle exit

L. Crozier, R. Foy, R. Adib, M. Badonyi, A. Kar, J. A. Holt, R. Wilson, C. Regnault, P. Whitfield, J. A. Marsh, A. T. Saurin, A. R. Barr, T. Ly

The paper shows that CDK4/6 inhibitor-induced G1 arrest causes cell overgrowth, triggering osmotic stress and p38-p53-p21 signaling to promote cell cycle exit.

bioRxiv · Sep 8, 2022Cell Biology