Author

C. Richichi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Organism:human· 1 Assay:metabolomics· 1 Organism:mouse· 1

Frequent Co-Authors

G. Pelicci· 2 shared A. Cavalli· 1 shared A. Fantin· 1 shared A. Ghisleni· 1 shared B. Balboni· 1 shared B. T. Ang· 1 shared C. Tacconi· 1 shared C. Tang· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersCancer Biology

G. Pelicci

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Metformin antiproliferative activity is exclusively mediated by the membrane functional expression of the Chloride Intracellular Channel 1 in glioblastoma stem cells

I. Verduci, F. Cianci, R. Cazzoli, G. Cannavale, M. Veronesi, B. Balboni, M. Ranucci, L. M. G. Palloni, C. Richichi, S. Faletti, D. Osti, G. Ailuno, G. Caviglioli, C. Tacconi, S. GIROTTO, , M. Mazzanti

This study identifies tmCLIC1 as the specific metformin receptor in glioblastoma stem cells, showing that metformin's antiproliferative effect depends on its binding to tmCLIC1 and subsequent inhibition of mitochondrial respiration.

bioRxiv · Dec 31, 2022 · PreprintCancer Biology

Adaptive mechanoproperties characterize glioblastoma fitness for invasion

P. Monzo, M. Crestani, Y. K. Chong, K. Hennig, A. Ghisleni, Q. Li, C. Richichi, P. Maiuri, M. Balland, M. P. Sheetz, G. Pelicci, B. T. Ang, C. Tang, N. C. Gauthier

The paper identifies FMN1 as a driver of glioblastoma invasiveness by linking it to increased cell stiffness, mechanical forces, and stochastic motility.

bioRxiv · Jun 17, 2020Cell Biology