Author

D. Attwell

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Assay:imaging· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:differential expression· 1 Assay:electrophysiology· 1

Frequent Co-Authors

C. Pearson· 1 shared D. Gill· 1 shared F. Freitas· 1 shared H. Sethi· 1 shared J. Rock· 1 shared N. Korte· 1 shared P. Singhal· 1 shared P. Tammaro· 1 shared

Activity by Year

2022-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperPhysiology

C. Pearson

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2 papers

Recent·Most cited

Pericyte-mediated constriction of renal capillaries evokes no-reflow and kidney injury following ischemia

F. Freitas, D. Attwell

This study identifies pericyte-mediated capillary constriction as a cause of no-reflow and kidney injury after ischemia, showing that targeting pericyte contractility can reduce injury.

eLife · Mar 14, 2022Peer-reviewedPathology

The Ca2+-gated Cl- channel TMEM16A amplifies capillary pericyte contraction reducing cerebral blood flow after ischemia

N. Korte, Z. Ilkan, C. Pearson, T. Pfeiffer, P. Singhal, J. Rock, H. Sethi, D. Gill, D. Attwell, P. Tammaro

This study shows that TMEM16A in pericytes amplifies capillary constriction and reduces cerebral blood flow after ischemia, suggesting it as a therapeutic target for stroke and related disorders.

bioRxiv · Feb 3, 2022 · PreprintPhysiology