Author

D. Bedja

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Pathology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:mouse· 1

Frequent Co-Authors

B. L. Lin· 1 shared C. C. Talbot· 1 shared C. Lukban· 1 shared D. Cihakova· 1 shared M. K. Wood· 1 shared M. V. Talor· 1 shared N. L. Diny· 1 shared N. Wang· 1 shared

Activity by Year

2022-2022 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 36 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperPathology

B. L. Lin

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

Hypereosinophilia causes progressive cardiac pathologies in mice

N. L. Diny, M. K. Wood, T. Won, M. V. Talor, C. Lukban, D. Bedja, N. Wang, C. C. Talbot, B. L. Lin, D. Cihakova

This study shows that hypereosinophilia in mice leads to cardiac inflammation and impaired cardiomyocyte contractility, contributing to heart dysfunction.

bioRxiv · May 4, 2022Pathology