Author

C. C. Talbot

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Evolutionary Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Approach:comparative / phylogenetic analysis· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

A. Cammarato· 1 shared A.-C. Wei· 1 shared B. L. Lin· 1 shared C. Lukban· 1 shared C. M. Antonescu· 1 shared D. Bedja· 1 shared D. Cihakova· 1 shared D. Mohr· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperEvolutionary Biology

A. Cammarato

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Author profile →

2 papers

Recent·Most cited

Hypereosinophilia causes progressive cardiac pathologies in mice

N. L. Diny, M. K. Wood, T. Won, M. V. Talor, C. Lukban, D. Bedja, N. Wang, C. C. Talbot, B. L. Lin, D. Cihakova

This study shows that hypereosinophilia in mice leads to cardiac inflammation and impaired cardiomyocyte contractility, contributing to heart dysfunction.

bioRxiv · May 4, 2022Pathology

CaMKII oxidation is a performance/disease trade-off in vertebrate evolution

Q. Wang, E. O. Hernandez-Ochoa, M. C. Viswanathan, I. D. Blum, J. M. Granger, K. R. Murphy, A.-C. Wei, S. Aja, N. Liu, C. M. Antonescu, L. D. Florea, C. C. Talbot, D. Mohr, K. R. Wagner, S. Regot, , M. E. Anderson

This study shows CaMKII oxidation by ROS evolved in vertebrates to enhance performance but increased disease risk.

bioRxiv · Sep 13, 2019Evolutionary Biology