Author

N. L. Diny

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Immunology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:bulk RNA-seq· 2 Research Focus:cell signaling & trafficking· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1

Frequent Co-Authors

B. Stockinger· 2 shared Y. Li· 2 shared A. Franchet· 1 shared A. Liebert· 1 shared A. P. Gould· 1 shared A. Sateriale· 1 shared B. L. Lin· 1 shared C. Alexandre· 1 shared

Activity by Year

2022-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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2 shared papersCancer Biology

B. Stockinger

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Drosophila AHR limits tumor growth and stem cell proliferation in the intestine

M. Tsai, J. Sun, C. Alexandre, M. Shapiro, A. Franchet, Y. Li, A. P. Gould, J.-P. Vincent, B. Stockinger, N. L. Diny

This study shows that Spineless, the Drosophila AHR homolog, limits tumor growth and stem cell proliferation in the adult intestine by regulating gene expression and metabolism.

bioRxiv · May 17, 2023 · PreprintCancer Biology

Dietary environmental factors shape the immune defence against infection with Cryptosporidium

M. R. Maradana, N. B. Marzook, O. Diaz Perez, T. Mkandawire, N. L. Diny, Y. Li, A. Liebert, K. Shah, M. Tolaini, M. Kvac, B. Stockinger, A. Sateriale

The paper shows that dietary metabolites enhance CD8+ T-cell immunity against Cryptosporidium infection in mice by activating the AHR pathway.

bioRxiv · Mar 30, 2023 · PreprintImmunology

Hypereosinophilia causes progressive cardiac pathologies in mice

N. L. Diny, M. K. Wood, T. Won, M. V. Talor, C. Lukban, D. Bedja, N. Wang, C. C. Talbot, B. L. Lin, D. Cihakova

This study shows that hypereosinophilia in mice leads to cardiac inflammation and impaired cardiomyocyte contractility, contributing to heart dysfunction.

bioRxiv · May 4, 2022Pathology