Author

D. Canetti

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Biochemistry· 1 Pathology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

A. Bonaud· 1 shared A. Carpinteiro· 1 shared A. Corazza· 1 shared A. Jaccard· 1 shared A. Lampis· 1 shared A. Rinsant· 1 shared A. Vanoli· 1 shared B. Foglia· 1 shared

Activity by Year

2019-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperPathology

A. Bonaud

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

A mouse model of cardiac AL amyloidosis unveils mechanisms of tissue accumulation and toxicity of amyloid fibrils

G. Martinez-Rivas, M. V. Ayala, S. Bender, G. R. Codo, K. W. Swiderska, A. Lampis, L. Pedroza, M. Merdanovic, P. Sicard, E. Pinault, L. Richard, F. Lavatelli, D. Canetti, A. Rinsant, K. Sihem, , C. Sirac

This paper develops a mouse model of cardiac AL amyloidosis that reproduces human disease features, including amyloid deposits and cardiac dysfunction, and identifies a role for partial LC degradation in fibril formation.

Nature Communications · Mar 27, 2025Peer-reviewedPathology

Amyloid damage to islet β-cells in type 2 diabetes: hypoxia or pseudo-hypoxia?

V. Bellotti, A. Corazza, B. Foglia, E. Novo, J. P. Simons, P. P. Mangione, G. Verona, D. Canetti, P. Nocerino, L. Obici, A. Vanoli, M. Paulli, S. Giorgetti, S. Raimondi, R. Al-Shawi, , M. Parola

This study investigates whether amyloid deposition in islet β-cells causes hypoxia or pseudo-hypoxia, contributing to type 2 diabetes pathogenesis.

bioRxiv · Oct 24, 2019 · PreprintBiochemistry