Author

D. Ewin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 2 Assay:metagenomics· 2 Organism:C. elegans· 1 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1 Research Focus:ecology & biodiversity· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

A. M. Buckley· 2 shared I. Moura· 2 shared M. Wilcox· 2 shared A. Buckley· 1 shared C. H. Chilton· 1 shared E. Clark· 1 shared G. Douce· 1 shared I. B. Moura· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersMicrobiology

A. M. Buckley

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

MiGut: a scalable in vitro platform for simulating the human gut microbiome - development, validation, and simulation of antibiotic induced dysbiosis

W. Davis Birch, I. Moura, D. Ewin, M. Wilcox, A. Buckley, P. Culmer, N. Kapur

MiGut is an in vitro platform that simulates the human gut microbiome and validates its response to antibiotics, showing good correlation with existing models.

bioRxiv · Sep 2, 2022Microbiology

Insights into the regulatory mechanisms of Clostridioides difficile biofilm formation

A. M. Buckley, D. Ewin, I. Moura, M. Wilcox, G. Douce

This study investigates the transcriptional changes and regulatory pathways in Clostridioides difficile during biofilm formation, identifying metabolic reprogramming and small RNAs involved in biofilm regulation.

bioRxiv · Feb 19, 2021 · PreprintMicrobiology

Profiling the effects of rifaximin on the healthy human colonic microbiota using a chemostat model

I. B. Moura, A. M. Buckley, D. Ewin, E. Clark, S. Mitra, M. H. Wilcox, C. H. Chilton

This study evaluates three rifaximin formulations on the human colonic microbiota using chemostat models, finding that formulation {kappa} caused the least disruption.

bioRxiv · Nov 1, 2019 · PreprintMicrobiology