Author

D. Khalili

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Immunology· 2

Signature Assays, Organisms & Methods

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Organism:Drosophila· 4 Approach:experimental assay· 4 Research Focus:cell signaling & trafficking· 3 Research Focus:development & morphogenesis· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:host-pathogen & immune response· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

U. Theopold· 4 shared M. Kunc· 2 shared A. M. Mueller· 1 shared C. Kalcher· 1 shared J. Ankarklev· 1 shared M. Mohammed· 1 shared R. Krautz· 1 shared S. Baumgartner· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 15 mutual co-author links

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4 shared papersCancer Biology

U. Theopold

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Single-cell sequencing of tumor-associated macrophages in a Drosophila model.

D. Khalili, M. Mohammed, M. Kunc, J. Ankarklev, U. Theopold

The paper uses a Drosophila model to study tumor-associated macrophages via single-cell RNA-sequencing, identifying distinct hemocyte clusters and their differential gene expression in tumor versus wild-type larvae.

bioRxiv · Jun 17, 2023 · PreprintCancer Biology

Chitinase-like proteins promoting tumorigenesis through disruption of cell polarity via enlarged endosomal vesicles.

D. Khalili, M. Kunc, S. Herbrich, A. M. Mueller, U. Theopold

This study identifies Idgf3, a chitinase-like protein, as promoting tumorigenesis in Drosophila by disrupting cell polarity through enlarged endosomal vesicles.

bioRxiv · Aug 17, 2022 · PreprintCancer Biology

Anti-fibrotic activity of an antimicrobial peptide in a Drosophila model.

D. Khalili, C. Kalcher, S. Baumgartner, U. Theopold

This study shows that overexpression of the Ras oncogene in Drosophila salivary glands causes fibrotic lesions, which are reduced by the antimicrobial peptide Drosomycin.

bioRxiv · Mar 19, 2021 · PreprintImmunology

Tissue-autonomous immune response regulates stress signalling during hypertrophy

R. Krautz, D. Khalili, U. Theopold

The paper investigates how a tissue-autonomous immune response modulates stress signaling during hypertrophy in Drosophila salivary glands, revealing a role for Drosomycin in inhibiting JNK signaling and preventing programmed cell death.

bioRxiv · Sep 30, 2019Immunology