Author

D. L. Marks

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Immunology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Organism:human· 1

Frequent Co-Authors

K. A. Michaelis· 2 shared K. G. Burfeind· 2 shared M. A. Norgard· 2 shared P. R. Levasseur· 2 shared X. Zhu· 2 shared A. A. Mangaonkar· 1 shared A. Buradkar· 1 shared A. Vedder· 1 shared

Activity by Year

2017-2019 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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2 shared papersImmunology

K. A. Michaelis

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

RAS mutations drive proliferative chronic myelomonocytic leukemia via activation of a novel KMT2A-PLK1 axis

R. M. Carr, D. Vorobyev, T. Lasho, D. L. Marks, E. J. Tolosa, A. Vedder, L. Almada, A. Yurchenko, I. Padioleau, B. Alver, G. Coltro, M. Binder, S. L. Safgren, I. Horn, X. You, , M. M. Patnaik

This study identifies RAS mutations as drivers of proliferative CMML through a novel KMT2A-PLK1 axis, showing PLK1 inhibition can improve disease outcomes in patient-derived xenografts.

bioRxiv · Dec 23, 2019 · PreprintCancer Biology

A distinct neutrophil population invades the central nervous system during pancreatic cancer

K. G. Burfeind, X. Zhu, M. A. Norgard, P. R. Levasseur, B. Olson, K. A. Michaelis, E. R. S. Torres, S. Jeng, S. McWeeney, J. Raber, D. L. Marks

This study identifies a novel neutrophil population invading the brain during pancreatic cancer, mediated by CCR2/CCL2 and P2RX7, leading to anorexia and muscle catabolism.

bioRxiv · Jun 3, 2019 · PreprintImmunology

TRIF is a key inflammatory mediator of acute sickness behavior and cancer cachexia

K. G. Burfeind, X. Zhu, P. R. Levasseur, K. A. Michaelis, M. A. Norgard, D. L. Marks

This study shows TRIF is crucial for inflammation-driven sickness behavior and cancer cachexia, with TRIFKO mice showing reduced symptoms and inflammatory markers.

bioRxiv · Dec 11, 2017Physiology