Author

D. Romashko

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:metagenomics· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

A. Flohr· 1 shared A. Mayweg· 1 shared A. Salomatov· 1 shared D. Epstein· 1 shared E. Buck· 1 shared J. Zhang· 1 shared M. O'Connor· 1 shared N. Ishiyama· 1 shared

Activity by Year

2019-2019 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCancer Biology

A. Flohr

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1 paper

Recent·Most cited

EGFR Oncogenes Expressed In Glioblastoma Are Activated As Covalent Dimers And Paradoxically Stimulated By Erlotinib

E. Buck, M. O'Connor, D. Romashko, A. Salomatov, N. Ishiyama, D. Epstein, A. Mayweg, A. Flohr, R. Iacone, T. Nicolaides, S. Markovic, J. Zhang

The paper identifies a mechanism by which extracellular domain EGFR mutations in glioblastoma are activated through covalent dimerization, rendering ATP-site inhibitors ineffective and paradoxically promoting proliferation.

bioRxiv · Oct 21, 2019 · PreprintCancer Biology